Abstract
α-Bungarotoxin (αBTX) administration in ovo prevents motoneuron apoptosis during development. This process may be mediated by α BTX-sensitive nicotinic cholinoceptors in the spinal cord, at the neuromuscular junction or at both sites. In order to differentiate between these possibilities, neuronal bungarotoxin binding (NBTX) binding to embryonic muscle and spinal cord was investigated in the chick.
Published Version
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