Abstract
Coronavirus disease (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS CoV-2) has turned out to be a formidable pandemic. Upcoming evidence from confirmed cases of COVID-19 suggests an anticipated incursion of patients with neurological manifestations in the weeks to come. An expression of the angiotensin-converting enzyme 2 (ACE 2), the cellular receptor for SARS-CoV-2 over the glial cells and neurons have made the brain a potential target. Neurotoxicity may occur as a result of direct, indirect and post-infectious complications. Attention to neurological deficits in COVID-19 is fundamental to ensure appropriate, timely, beneficial management of the affected patients. Most common neurological manifestations seen include dizziness, headache, impaired consciousness, acute cerebrovascular disease, ataxia, and seizures. Anosmia and ageusia have recently been hinted as significant early symptoms in COVID-19. As cases with neurological deficits in COVID-19 emerge, the overall prognosis is yet unknown.
Highlights
Neurological symptoms of coronavirus disease (COVID19) can be minimal at presentation but these can cause monumental damage as silent contagious sources
There is a compelling need to understand the potential of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) to cause neurological illness for proper diagnosis and therapeutic decision making
Neurological symptoms fall into three main categories: central nervous system (CNS), peripheral nervous symptoms (PNS) and skeletal muscular symptoms.[3]
Summary
Follow this and additional works at: https://ecommons.aku.edu/pakistan_fhs_mc_med_neurol Part of the Life Sciences Commons, Neurology Commons, and the Virus Diseases Commons. The Journal of the Pakistan Medical Association, 70(5(Suppl 3)), S101-S103.
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