Abstract

In recent years, neurobiology has produced the cellular foundations that account for the phenomena of memory and learning in mammals. Nevertheless, this information has not always been applied to educational processes. Our paper reviews the information regarding the cellular processes underlying learning and how these may impact or explain didactic processes, forcing us to rethink current paradigms.

Highlights

  • In the course of his or her duties, the professor routinely faces a series of problems and challenges with the aim of effecting true learning on the part of his students

  • Upon realizing a search to define learning, we encountered two main currents of thought, one relating to the fields of psychology and sociology, which understood learning as a change in conduct of a permanent nature in a student, especially associated to environmental stimuli

  • The majority of the areas studied within the hippocampus are located in CA1, and the different types of protocols involved indicate the complexity of explaining the processes that occur in Long term potentiation (LTP) and long term depression (LTD)

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Summary

Introduction

In the course of his or her duties, the professor routinely faces a series of problems and challenges with the aim of effecting true learning on the part of his students. Upon realizing a search to define learning, we encountered two main currents of thought, one relating to the fields of psychology and sociology, which understood learning as a change in conduct of a permanent nature in a student, especially associated to environmental stimuli. It is precisely this vision that one begins to internalize and use, beginning with entry into the pedagogical field—leaving aside, and even behind permanently, the biological elements that influence learning. We focus the present work for having more discussion of the cells model and the child complex education process and in the future, working more close both discipline

The Contribution of Neurobiology
Calcium as a Secondary Messenger
Signalling through G Protein Coupled Receptors
Synaptic Metaplasticity
Long Term Potentiation and Long Term Depression
Conclusion
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