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Abstract
Abstract A single bout of exercise can decrease blood pressure level in hypertensive individuals and this phenomenon is known as post-exercise hypotension (PEH). PEH is clinically important and reduces blood pressure after physical exercise in hypertensive subjects. This reduction has been attributed to autonomic mechanisms, e.g., reduced peripheral sympathetic activity, adjustments in cardiac autonomic balance and baroreflex sensitivity. Besides, evidence has suggested that the central baroreflex pathway has an important role in the occurrence of PEH. Therefore, the aim of this study was to review the effects of physical exercise on areas of the central nervous system involved in the regulation of blood pressure.
Highlights
Systemic hypertension is a multifactorial clinical condition and an independent risk factor for mortality in patients with cardiovascular diseases[1]
This review emphasizes on evidence of synaptic mechanisms in the central baroreflex pathway that contribute to development of post-exercise hypotension (PEH)
Chen et al.[71] in an elegant study confirmed that microinjection of a substance P–NK1-R antagonist in the nucleus tractus solitarii (NTS) immediately before exercise attenuates the development of PEH in spontaneously hypertensive rats (SHRs)
Summary
Systemic hypertension is a multifactorial clinical condition and an independent risk factor for mortality in patients with cardiovascular diseases[1]. Post-exercise hypotension [PEH] is a prolonged decrease in arterial blood pressure after a single bout of exercise[7].
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