Year-end Sale % OFF 
Abstract
Cessation of breathing and cyclic variations in tidal volume and frequency are far more likely to be precipitated when hypoxia or transient hyperventilation produces subnormal levels of PCO2 in NREM sleep and at sleep onset, than in awake subjects. The ability of the awake subject to breathe with only rare apneas and oscillations in ventilation at levels of PCO2 considerably below resting seems to depend on the interplay of chemical and neural drives.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
