Abstract
AbstractRecently, Vamos et al. (1981) reported that the clinical features in the neonatal onset form of multiple sulphatase deficiency (MSD) were much more pro nounced than those of the usual type of MSD. The activities of arylsulphatases A, B and C in the patient's cultured skin fibroblasts were greatly reduced. This paper characterizes the biochemical features of neonatal MSD and distinguishes this disorder from typical MSD.
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