Abstract
The synthesis of NO in endothelial cells plays a major role in the control of vascular homeostasis in part due to its ability to inhibit vascular tone and activation of platelets. Inflammatory mediators such as IL-1β impair the NO-mediated endothelium-dependent relaxations in isolated blood vessels. Since IL-1β is a potent inducer of the inducible NO synthase (iNOS), the role of the induced synthesis of NO and the inhibitory effect of IL-1β was examined.
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