Abstract

Summary Left posterior subdivision block (LPSB) diminishes or masks the ECG-VCG signs of a necrotic area of 2.5 X 3 cm produced in the posterior aspect of the free left ventricular wall of the dog's heart. This was observed in subepicardial necrosis, as well as in subendocardial, intramural or transmural. In cases of subendocardial necrosis, the determination of the activation times in the preserved subepicardial muscular layer demonstrated a moderate delay, interpreted as due to peri-infarction block, which did not give rise to outstanding manifestations in peripheral leads. When a LPSB was associated, the activation of this region was considerably delayed and important ECG changes took place in peripheral leads. Even in the presence of necrosis, the signs of LPSB were clearly recognized in both the ECG-VCG curves.

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