Abstract
Background and Aims : Neutrophil extracellular traps (NETs) have emerged as important contributors to thrombus formation in acute myocardial infarction (AMI) fueling the pro-thrombotic milieu. Obtaining insights on inducers and inhibitors of NETosis is of high importance. We have previously demonstrated that a subset of extracellular vesicles (EV) carrying oxidation specific epitopes (OSE), products of lipid peroxidation, is elevated in AMI. Importantly, natural IgM antibodies targeting OSE are considered for their protective role in cardiovascular disease.
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