Abstract
High-density lipoprotein (HDL) has been reported to have cardioprotective properties independent from its cholesterol transport activity. We have investigated modulating the composition of reconstituted HDL (rHDL) as a means of expanding its cardioprotective effects. The influence of rHDL and native HDL on Stat3, the transcription factor playing an important role in myocardium adaptation to stress, was analyzed in ventricular cardiomyocytes.
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