Abstract
To evaluate the effect of membranous lesions in the etiology of sleep apnea syndrome (SAS), nasal resistance (NR) was investigated before and 15 min after 0.02% epinephrine was sprayed into the nasal cavity in 14 patients with SAS. NR was detected by the active anterior method in a sitting position. In five patients, NR was elevated beyond the measurable range. NR was increased in the remaining 9 patients as compared with values in a control group; however, the difference between the two groups was not statistically significant. Epinephrine spray reduced NR to a greater degree in patients with SAS than in healthy controls. Membranous hypertrophy in the turbinates may be greater in SAS patients than in healthy individuals.
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