Abstract
On the effect of ethanol administration on porphyrin metabolism in rats intoxicated with lead acetate, the urinary excretion of δ-aminolevulinic acid and coproporphyrin was determined, and the activity of δ-aminolevulinic acid produced in rats liver homogenate was measured. The following results were obtained.1) The urinary excretion of coproporphyrin and δ-aminolevulinic acid in rats intoxicated with lead acetate was increased by ethanol treatment.2) It was demonstrated that the hepatic δ-aminolevulinic acid producing activity in rats intoxicated with lead acetate was additively enhanced by ethanol treatment.3) The activation of hepatic δ-aminolevulinic acid producing activity with ethanol was inhibted by pre-treatment of pyrazole.
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