Abstract

The presence of myocardial fat is not a finding that is infrequent during postmortem examinations. The case under discussion showed an extensive amount of fat, markedly on the right and mildly on the left ventricles. The differentiation of physiological fat from pathological entities is essential. In this case, two entities were initially suspected namely Arrhythmogenic Right Ventricular Cardiomyopathy/Dysplasia and excessive physiological fat deposition of the heart. A 66-year-old averagely build, a mentally subnormal, previously healthy female was found dead after a nonspecific, mild, general ill-health. Trauma and the possibility of poisoning were excluded. The cause of death was concluded as coronary artery disease with further investigations. The incidental finding of an excessive amount of fat as seen in this case warrants explanations and future research in Sri Lanka.

Highlights

  • Research interests have been increased to study the relationship between the amounts of fatty infiltration of the heart in myocardial diseases

  • The first, is the exclusion of any pathological fat deposition of her heart and the is the exclusion of any possible contribution or causation to cause death by that finding

  • The cause of death was kept under investigation, and with the investigative results, the cause of death was attributed to coronary artery disease ascribing the excessive cardiac fat as physiological or due to aging

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Summary

Introduction

Research interests have been increased to study the relationship between the amounts of fatty infiltration of the heart in myocardial diseases. The first, is the exclusion of any pathological fat deposition of her heart and the is the exclusion of any possible contribution or causation to cause death by that finding. The myocardium shows apparent fatty tissue even in trabeculae (Fig.). No history of past episodes of IHD, diabetes mellitus or hypertension or any other metabolic diseases was noted. She was physically fit excluding muscular dystrophies. The right ventricle microscopically showed fibrofatty replacement with intervening unaffected myocytes with a thick epicardial fat layer. The left ventricle showed fatty infiltration within the myocardium without any inflammatory cells. The cause of death was kept under investigation, and with the investigative results, the cause of death was attributed to coronary artery disease ascribing the excessive cardiac fat as physiological or due to aging

Discussion
Findings
Conclusions
Arrhythmogenic
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