Abstract
Mushroom poisoning is common in the United States. The severity of mushroom poisoning may vary, depending on the geographic location, the amount of toxin delivered, and the genetic characteristics of the mushroom. Though they could have varied presentation, early identification with careful history could be helpful in triage. We present a case of a 69-year-old female of false morel mushroom poisoning leading to hepatotoxicity with painless jaundice and biochemical pancreatitis.
Highlights
BackgroundMushroom poisoning is common in the United States with 6000 exposures annually [1]
Seasonal variation might help in predicting the type of poisoning—Amanita species occurring in fall and Gyromitra species in spring and summer
A probable diagnosis of mushroom poisoning was made with history, symptoms and exclusion of other causes
Summary
Mushroom poisoning is common in the United States with 6000 exposures annually [1]. In some regions (the Rocky Mountain region and the Pacific Northwest) the reporting is quite extensive [2]. She reported to the hospital with nausea, vomiting and painless jaundice. Obese woman with jaundice, lower extremity pitting edema Her vitals were significant for tachycardia (rate > 120 beats/min) with AF, blood pressure, respirations and temperature was within normal limits. Her complete blood count (CBC) with differential was unremarkable Her creatinine was elevated at 1.5 mg/dl (baseline < 1). Her bilirubin peaked at 16 mg/dl and trended down rapidly in two days and was 6.9 mg/dl during discharge; aspartate transaminase (AST) and alanine transaminase (ALT) were trending down On further questioning, she acknowledges to consuming wild mushroom (looks like morels) hunted and given by his son in the beginning of summer, a day before presentation. A probable diagnosis of mushroom poisoning was made with history, symptoms and exclusion of other causes
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