Abstract
Background and Aims : Diabetes Mellitus (DM) is a metabolic disorder affecting ten percent of the worldwide population. The morbidity of DM patients is strongly correlated to cardiovascular diseases (CVD), including cerebrovascular disease, peripheral vascular conditions, and ischemic heart disease. Previous studies have shown that vascular smooth muscle cells (VSMC) from DM patients are often functionally impaired with increased expressions of inflammatory and pro-fibrotic genes. Given its importance in arteriopathy, we aim to understand better the molecular pathway leading to VSMC phenotype modulation in DM.
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