Multiple Sclerosis Coupled with Ataxia, Muscle Atrophy, Dysautonomia and Anhydrosis: A Functional Neurology Approach

Abstract

Event Abstract Back to Event Multiple Sclerosis Coupled with Ataxia, Muscle Atrophy, Dysautonomia and Anhydrosis: A Functional Neurology Approach Gil S. Jaudy1* 1 Jaudy Treatment Center, United States Background and Aims A 56-year-old male, presented to our office complaining of insidious neurological dysfunctions that started on 10, 1991. He reported consistent numbness and inability to walk without assistant device since 2009. He also complained of neck pain and low back pain on a scale of 8/10 (10 being the worst pain). History included right shoulder dislocation, whiplash, mononucleosis, bad reaction to Swine Flu Vaccine in 1977, MVA in 1983 which ended plastic surgery to his forehead, bad reaction to Hepatitis B Vaccine in 1988, Diagnosed with parasites and Lyme disease in 1989. 1990 he experienced the first episode of anhydrosis. 1992 was diagnosed with Multiple Sclerosis and was administered oral steroid, prednisone and Copaxone. 2010 he had exacerbation of Multiple Sclerosis symptoms. Advanced radiology brain MRI 09/22/2006 described new lesions since the previous study, dated 06/26/2002. The report stated progression of callosal volume loss and cerebral atrophy. Methods Upon examination, on 04/19/2011, spontaneous legs movement was observed. Opthalmoscopic examination showed V:A ratio 3:1 on the right. OPK showed abnormal upward to downward saccades ratio. Horizontal saccades preserved. Irregular pursuits, and square wave jerks were observed. Right soft palate paresis was observed. Left facial weakness. Cardiac auscultation split S2. Positive convergence and cardinal fields. Romberg’s stance with severe sway. Bilateral positive Babinski sign and jerky myoclonic activity. Paresis of left big toe. Bilateral dysmetria. Positive Myerson’s sign. Bilateral decreased DTR’s. Bilateral muscle spasticity in the lower extremity and hypotonia in the upper extremity. Cortical blind spot showed right partial hemianopia. Treatment consisted of Brain-Based therapeutic activities, segmental coupled motion reduction, myofascial release, vestibular stimulation, timed breathing exercises, complex movement activities, visual stimulation, sound stimulation, SEP, activities of daily living, and nutritional consultation. Results Patient showed progressive improvement in his motor function and decrease in muscle spasticity. Pathological reflexes were resolved. He showed improvement in gait and reported that he was able to take couple of steps without assistance. He reported ability to move his left big toe after he had lost this function for more than 20 years. A laboratory test obtained on 04/21/2012 by his primary showed no signs of Multiple Sclerosis. Acknowledgements Abstracts are clinically documented by the Jaudy Treatment Center. All clinical data is available upon request. Jaudy Treatment Center (JTC), all rights reserved, 2016. References Jaudy Treatment Center clinical data, 2016. Keywords: multiple sclerosis (MS), Ataxia, muscle atrophy, dysautonomia, Anhydrosis Conference: International Symposium on Clinical Neuroscience: Clinical Neuroscience for Optimization of Human Function, Orlando, United States, 7 Oct - 9 Oct, 2016. Presentation Type: Poster Presentation Topic: Abstracts ISCN 2016 Citation: Jaudy GS (2016). Multiple Sclerosis Coupled with Ataxia, Muscle Atrophy, Dysautonomia and Anhydrosis: A Functional Neurology Approach. Front. Neurol. Conference Abstract: International Symposium on Clinical Neuroscience: Clinical Neuroscience for Optimization of Human Function. doi: 10.3389/conf.fneur.2016.59.00086 Copyright: The abstracts in this collection have not been subject to any Frontiers peer review or checks, and are not endorsed by Frontiers. They are made available through the Frontiers publishing platform as a service to conference organizers and presenters. The copyright in the individual abstracts is owned by the author of each abstract or his/her employer unless otherwise stated. Each abstract, as well as the collection of abstracts, are published under a Creative Commons CC-BY 4.0 (attribution) licence (https://creativecommons.org/licenses/by/4.0/) and may thus be reproduced, translated, adapted and be the subject of derivative works provided the authors and Frontiers are attributed. For Frontiers’ terms and conditions please see https://www.frontiersin.org/legal/terms-and-conditions. Received: 02 Sep 2016; Published Online: 07 Sep 2016. * Correspondence: Dr. Gil S Jaudy, Jaudy Treatment Center, Palm Desert, CA, United States, drjaudy@hotmail.com Login Required This action requires you to be registered with Frontiers and logged in. To register or login click here. Abstract Info Abstract The Authors in Frontiers Gil S Jaudy Google Gil S Jaudy Google Scholar Gil S Jaudy PubMed Gil S Jaudy Related Article in Frontiers Google Scholar PubMed Abstract Close Back to top Javascript is disabled. Please enable Javascript in your browser settings in order to see all the content on this page.