Abstract
AbstractDiabetes is associated with numerous neuropathies; some accompanies poor glycemic control including diabetic ketoacidosis‐associated (DKA) neuropathies, others occur following improved glycemic control including treatment‐induced neuropathies of diabetes (TIND). A 27‐year‐old poorly controlled type I diabetic female developed multifocal weakness and pains accompanied by widespread autonomic dysfunction following an episode of DKA with rapid and subsequent sustained glycemic control. Injury was thought to have occurred secondary to disruptions of the endoneurial microenvironment following rapid glycemic control.
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