Abstract
We analyzed group B Streptococcus (GBS) neonatal invasive infections reported during 2007–2019 in France. The hypervirulent clonal complex (CC) 17 GBS was responsible for 66% (827/1,262) of cases. The role of CC17 GBS increased over time (p for trend = 0.0001), together with the emergence of a multidrug-resistant CC17 GBS sublineage.
Highlights
We analyzed group B Streptococcus (GBS) neonatal invasive infections reported during 2007–2019 in France
Two GBS-associated syndromes are distinguished in neonates: early-onset disease (EOD), which occurs during the first week of life, and late-onset disease (LOD), which occurs after the first week [1]
The proportions of EOD and LOD and the associated clinical manifestations described here are very close to the national estimations
Summary
We analyzed group B Streptococcus (GBS) neonatal invasive infections reported during 2007–2019 in France. We analyzed neonatal invasive GBS diseases reported to the French National Reference Center for Streptococci during 2007–2019 and investigated the role of the hypervirulent clone over this period. A total of 1,262 neonatal invasive infections (EOD, n = 394, 31%; LOD, n = 868, 69%) were reported during 2007–2019.
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