Abstract
Regulation of mRNA stability and translation is increasingly recognized as a critical control point in dynamically altering gene expression during stress or disease. In the heart, post‐transcriptional alteration of the level of select mRNAs has emerged as a driver of disease. Cardiac inflammation, hypertrophy, metabolism and electrophysiology are all aspects that are perturbed in pathophysiologic conditions and undergo regulation by RNA‐binding proteins. My talk will focus on novel molecular mechanisms responsible for controlling cardiac mRNA fate in heart disease.
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