Abstract
It has been suggested that mutation of FGFR3 is associated with non-invasive tumors of low malignant potential and low risk of recurrence and progression. We hypothesize that FGFR3-mutated tumors are more genetically stable than bladder tumors without mutation. The aim of this study was to analyze the distribution of FGFR3 mutations in bladder tumors of different grade and stage and determine the relation of FGFR3 mutations to chromosomal alterations detected by CGH.
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