Abstract
Diving in cold water (15 °C) results in abrupt cessation of intercostal inspiratory muscle activity, whereas expiratory muscle activity is enhanced. This response is evoked by stimulation of cold thermoreceptors located in the bill's skin since it is delayed when water temperature is 37 °C. Very weak mechanical stimulations of the glottis and nostril regions resulted in a strong closure of the glottis. The role of the thermoreceptors of the bill's skin and of the glottis mechanoreceptors is discussed in relation to the triggering mechanism of diving apnoea.
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