Abstract

We describe a case of a 37-year-old man who developed prolonged papilledema as a result of increased intracranial pressure following unilateral radical neck dissection due to diffuse large B-cell non-Hodgkin lymphoma. It seems that the increase in intracranial pressure was a result of insufficient collateral venous drainage of the brain. Clinical examination showed bilateral papilloedema, diplopia, and visual deterioration in the left eye. Magnetic resonance imaging and computed tomography of the brain were normal. Semi-automated kinetic perimetry was used to mo­nitor the visual function during 12 months of the follow-up. This method revealed enlargement of the blind spot in the left eye without progression. Peripheral borders of the visual field were within normal limits.

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