Abstract
Membranoproliferative glomerulonephritis has a broad possible range of causes. The syndrome of proliferative glomerulonephritis with monoclonal immunoglobulin deposits (PGNMID) as part of the monoclonal gammopathy of renal significance (MGRS) spectrum is not yet clearly defined; also unclear is the ability for low viral load, untreated hepatitis B to cause proliferative glomerulonephritis (GN) or the significance of anti-Ro in absence of other biochemical, histological or clinical classical lupus manifestations.
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