Abstract
Prestin, the electromotive protein found in outer hair cells (OHCs), is responsible for sound amplification without which the hearing system is dysfunctional. Known as an “incomplete transporter”, prestin transits between an expanded and a contracted state in response to membrane potential shifts, resulting in sudden alteration of the surface area in OHCs. Although prestin was discovered decades ago, its molecular mechanism and unique interactions with lipids to control the surface area in OHCs remained elusive until recently.
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