Abstract
The emergence and spread of carbapenem-resistant Enterobacteriaceae represents a major clinical and public health challenge, especially concerning those organisms that produce Klebsiella pneumoniae carbapenemase (KPC)–type b-lactamase. In this study, we examined the molecular epidemiology of KPC-producing Klebsiella pneumoniae strains, isolated during sporadic or epidemic cases in different hospitals of North-Western Tuscany (NWT).
Highlights
Introduction / objectives The emergence and spread of carbapenem-resistant Enterobacteriaceae represents a major clinical and public health challenge, especially concerning those organisms that produce Klebsiella pneumoniae carbapenemase (KPC)–type b-lactamase. In this study, we examined the molecular epidemiology of KPC-producing Klebsiella pneumoniae strains, isolated during sporadic or epidemic cases in different hospitals of North-Western Tuscany (NWT)
The PFGE method allowed to identify 7 variants clonally related (pt A1A7), sharing the same MLST sequence type (ST 258)
Summary
A Buzzigoli1*, P Valentini[1,2], M Minacori[1], F Guarneri[1,2], R Banducci[1,2], C Tascini[3], F Menichetti[3], GM Rossolini[4], B Casini[1,5], GP Privitera[1,2]. From International Conference on Prevention & Infection Control (ICPIC 2011) Geneva, Switzerland. Introduction / objectives The emergence and spread of carbapenem-resistant Enterobacteriaceae represents a major clinical and public health challenge, especially concerning those organisms that produce Klebsiella pneumoniae carbapenemase (KPC)–type b-lactamase. In this study, we examined the molecular epidemiology of KPC-producing Klebsiella pneumoniae strains, isolated during sporadic or epidemic cases in different hospitals of North-Western Tuscany (NWT)
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