Abstract

A Turkish infant of 96 days is accepted to the pediatric emergency room after being carried by their parents because of inconsolable crying, persistent cough, and difficulty in feeding during the previous day. Although she was aware and nor signs nor symptoms was suggestive of an impending MOF, in a few minutes it takes over an asystolic cardiac arrest. It was found the exitus after 75 minutes of resuscitation. The autopsy report indicates as pathological cause of death a rare form of childhood idiopathic calcific arterial disease caused by mutation of the gene encoding the same name EPP1 enzyme.

Highlights

  • Clinical CaseThis case report aims to share with readers the experience of the medical team of the hospital A

  • A Turkish infant of 96 days is accepted to the pediatric emergency room after being carried by their parents because of inconsolable crying, persistent cough, and difficulty in feeding during the previous day

  • The review showed that the pathology is corresponding to very limited circumstances described in the literature, [6] which presents histological and microscopic similar features: widespread calcification blood vessels that it causes extensive hemorrhagic-necrotic areas hesitate within a framework of multi- organ failure irreversible

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Summary

Clinical Case

This case report aims to share with readers the experience of the medical team of the hospital A. Peripheral venous access, the anesthetist and the neonatologist were contacted to collaborate in assisting the infant [3,4]. On their arrival the general conditions were already deteriorating. She was immediately and successfully intubated by nasotracheal way; after a second way of intraosseous was taken, we begun resuscitation as guidelines. After 75 minutes of resuscitation, during which there has been no resumption of shockable rhythm, it finds death After three days it runs autopsy, requesting confirmation for alleged and suspected heart disease.

Discussion
Take Home Messages of the Clinical Case
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