Abstract
Vitamin E has been demonstrated to modulate systems. The present study shows that oral treatment of vitamin E (100 mg/kg for 6 days) can modulate some killing functions of alveolar macrophages (AMs) being important for lung defense. Vitamin E enhanced not only phagocytosis of opsonized particles by AMs but also active oxygen generation by opsonized zymosan (OZ) or concanavalin A (ConA)stimulated AMs. Nitrite production by OZ or Con Astimulated AMs showed a little increse by vitamin E treatment. However, interleukin-1 production by lipopolysaccharide-activated AMs was not different between control and vitamin E-treated groups. These results suggest that vitamin E can enhance some killing functions of AMs being important for lung defense.
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