Abstract
The model developed by Lekic and his colleagues provides a framework for a number of important studies on cerebellar hemorrhage. Using biochemical, histological and behavioral approaches they have mapped out the essential features of the model. Future directions will need to refine the behavioral studies to determine the sites in the cerebellum responsible for the behavioral effects. Are the biochemical changes produced by hemorrhage in cerebellar tissues similar to those in the basal ganglia? Will drugs that have a positive effect on supratentorial hemorrhage also work in the cerebellum? Do larger lesions produce brainstem compression with hydrocephalus, which would more closely model the acute illness in man? If so, can medical treatments reduce the secondary damage in such a model and prevent the mass effects that are now treated by surgical excision? This interesting report of a new model will stimulate interest in this important cause of stroke.
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