Abstract
SNEDDON SYNDROME (SNS) is a hypercoagulable state associated with ischemic cerebrovascular events and livedo reticularis.1 Other organ manifestations are heart valve disease and renal insufficiency.2 Although the underlying mechanism is poorly understood, the presence of antiphospholipid (APL) or anticardiolipin (ACL) antibodies may cause thrombotic events and valvulopathy.3 Irregular thickening of the mitral and/or aortic valve leads to valvular dysfunction and eventually stenosis, or most frequently, insufficiency.
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