Abstract
Mitofusin 2 (Mfn2) is a critical negative regulator of vascular smooth muscle cell (VSMC) hypertrophy and proliferation by regulating mitochondrial fusion, ras/raf/ERK signal transduction, oxidative stress, calcium infusion, and apoptosis [1,2]. The purpose of this study was to investigate if Mfn2 was related with aortic remodeling and the potential mechanism.
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