Abstract
CLPP (caseinolytic peptidase P) mediates degradation of unfolded mitochondrial proteins and activates mitochondrial unfolded protein response (mt-UPR) to maintain homeostasis in the presence of cellular and metabolic stress. The aim of the current study was to determine the role of mt-UPR in female reproductive competence and senescence using a mouse model with global germline ClpP deletion.
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