Abstract
The intestinal mucosal-arterial PCO2 (ΔPCO2) remains remarkably stable in anemic hypoxia suggesting that the villi perfusion is well-maintained1. The microcirculation, however, has been insufficiently studied in extreme hemodilution.
Highlights
The intestinal mucosal-arterial PCO2 (ΔPCO2) remains remarkably stable in anemic hypoxia suggesting that the villi perfusion is well-maintained1
Conclusions intestinal serosal and mucosal microcirculations were severely compromised in anemic hypoxia, the ΔPCO2 did not increase
The lack of change in ΔPCO2 cannot be ascribed to the preservation of villi perfusion
Summary
The intestinal mucosal-arterial PCO2 (ΔPCO2) remains remarkably stable in anemic hypoxia suggesting that the villi perfusion is well-maintained1. Microcirculatory alterations are more severe in anemic than in ischemic hypoxia G Ferrara1*, VS Kanoore Edul1, E Martins1, HS Canales1, C Canullán1, G Murias1, MO Pozo1, C Ince2, A Dubin1* Introduction The intestinal mucosal-arterial PCO2 (ΔPCO2) remains remarkably stable in anemic hypoxia suggesting that the villi perfusion is well-maintained1.
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