Abstract
MHRT expression during remote ischemic preconditioning in patients with coronary artery disease
Highlights
Remote ischemic preconditioning (RIPC) is a tissues or organs
We suggest that the change in the expression of long noncoding RNAs (lncRNAs) MHRT in this case may be an additional indicator for the RIPC effect on cardiovascular system
The stroke volume index (SVI) index in patients of the RIPC group was higher by 14 % and the rate of systemic vascular resistance (SVRI) was significantly lower in RIPC group
Summary
Remote ischemic preconditioning (RIPC) is a tissues or organs. Protective effects of RIPC short episode of ischemia-reperfusion of a are explained by releasing biochemical mescertain organ that may be protective for distant sengers that have protective properties. I. Drevytska, McClanahan et al for the first time demonstrated the protective effect of RIPC on myocardium [1]. McClanahan et al for the first time demonstrated the protective effect of RIPC on myocardium [1] They found that short periods of ischemia followed by reperfusion in kidney protected myocardium from prolonged ischemia and reduced the infarct size. Skeletal limb preconditioning demonstrates the beneficial effect for myocardium and endothelial tissues in patients [2]. There are a few studies showing that limb RIPC reduces cardiac, but neuronal injury after prolonged ische mia or cardiac arrest [3,4,5,6]
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