Methamphetamine Induced Cardiomyopathy

Younes I,Posimreddy S,Elkattawy S,Noori M

doi:10.36502/2020/hcr.6165

Abstract

Unfortunately, 35 million people worldwide suffer from drug use disorder while only one in seven people receive treatment. The health impacts combined with the socioeconomic burden of drug abuse are too numerous to count. It is well known that all organ systems are adversely affected by drug use including but not limited to the cardiovascular, respiratory, neurological, and renal. We will focus our attention on the effects of Methamphetamines on the cardiovascular system. Methamphetamines are known to be highly addictive stimulants with significant cardiovascular implications. We have gathered information from the literature available on methamphetamine-associated cardiomyopathy (MACM) and will discuss a case of a 58-year-old male, with no past medical history, who presented with dyspnea secondary to MACM.

Highlights

The socioeconomic burdens of drug abuse are immeasurable
A study showed that cardiovascular disease was found to be the most common contributing cause of death among methamphetamine users [3]
The cardiovascular complications of methamphetamine vary from acute life-threatening complications such as malignant hypertension, coronary vasospasm, acute myocardial

Summary

Introduction

The socioeconomic burdens of drug abuse are immeasurable. According to the United Nations World. The patient denied any dyspnea at rest but reported dyspnea on minimal exertion He reported weekly use of amphetamine for the last 10 years and >20 pack year smoking history; he denied any alcohol. Review of systems was negative for any recent chest pain, palpitations, productive cough, recent upper respiratory tract infection, fever, chills, or recent travel His family history was insignificant for heart or lung diseases. Disease with 50% stenosis proximal to mid LAD and ostial left circumflex artery (Fig-2). His vitals on admission were stable with a blood pressure of 130/93, heart rate of 108, respiratory rate of 18 and O2 saturation 98% on room air at rest. Fig-1: Echo Severely decreased global left ventricular systolic function; elevated left atrial and left ventricular end-diastolic pressures

Discussion

Findings

Conclusions