Abstract
Metabolic syndrome is reportedly one of the key health concerns worldwide. It is defined as a group of conditions including hypertension, dysglycemia, and abdominal obesity. The wear and tear of telomeres is known to be a major incident not only in mammalian aging, but also in distressed nutrient sensing, which may contribute to a number of metabolic dysfunctions. The metabolic syndrome was linked to the growing prevalence of obesity, which is at rise invariably in all age groups including elderly. The existing literature review focuses on the relationship of shorting of telomere and metabolic syndrome. As the shortening of telomeres influence cellular senescence and eventual stoppage of cell division.
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