Abstract
Metabolic Consequence of Short Term Starvation in Gastrointestinal Diseases: A Mini Review
Highlights
Effects of starvation were mainly evident on the degeneration of cells in digestive organs as seen in the shrinkage and separation of cells [1]
The combined treatment profoundly impairs cancer glucose metabolism and virtually abolishes lesion growth in experimental models of breast and colon carcinoma suggest that energy metabolism (EM) is more promising target to reduce cancer progression [3]
Short term starvation is the medical condition of congenital hypertrophic pyloric stenosis
Summary
The metabolic consequences of preoperative starvation and carbohydrate loading and mechanisms linking insulin resistance to impaired mitochondrial function are discussed. We just find out the short-term energy deprivation combinations with the health control subject. Followed by the nutrition and metabolism on molecular basis of per operative surgery. Majority of animal studies have shown that short-term energy deprivation decreases mitochondrial ATP synthesis capacity and complex activity, and increases oxidative injury which lead to development of insulin resistance (IR) during starvation and associated with an impaired mitochondrial function. From animal to human subject we thoroughly find out that short-term starvation (STS) causes mitochondrial dysfunction and it underlies the development of insulin resistance (IR) in patients undergoing elective surgery (ES)
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