Abstract
Metabolic bone disease is commonly seen in captive reptiles, especially Lizards and Chelonia. The reason for this lies with their mechanisms of Vitamin D and calcium metabolism. Many species do not appear to have the capability to absorb dietary Vitamin D. Instead they rely on the biosynthesis of endogenous vitamin D when in the presence of UV-B light and at a suitable temperature. Calcetriol (active Vitamin D) is required for absorption of dietary calcium. Therefore a lack of suitable UV-B provision, suitable temperature and suitable dietary calcium can all predispose to metabolic bone disease.
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