Abstract
Offspring born following fetal growth restriction (FGR) have an increased risk of cardiovascular disease. Chronic fetal hypoxaemia caused by placental insufficiency is the primary cause of FGR. Hypoxaemia is associated with increased oxidative stress in the fetus and subsequently reduces nitric oxide (NO) bioavailability. Antenatal melatonin therapy may prevent damage to the cardiovascular system, via antioxidant actions, and improve NO bioavailability.
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