Abstract
Use of sunlight as a therapeutic tool dates back as far as Hippocrates. In the modern era hundreds of articles have been published describing clinical and cellular responses to light. Photon therapy has demonstrated potent in vivo efficacy for treatment of ischemic conditions of acute myocardial infraction and stroke in multiple validated animal models. These results have been reproduced at independent academic centers. This paper will summarize what is understood about the mechanism of photon therapy. The effects of photon therapy are wavelength specific upon a known mitochondrial receptor (cytochrome C oxidase). Targeting of this receptor results in formation of adenosine triphosphate (ATP), enhanced mitochondrial survival and maintenance of cytochrome C oxidase activity (Eells et al., 2003; PhotoThera Scientific Report, 2003).
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