Abstract
Burst stimulation of the vagus nerve in cats results in synchronization of vagal and cardiac rhythms, while their desynchronization leads to sinus arrhythmia. Using a coefficient characterizing the magnitude of chronotropic effect, we have determined a zone of cardiocycle where the stimulating effect of the vagus stimulus was several times higher than in other zones. Somatostatin reduces this zone and increases the range of theP-P interval fluctuations in it, while somatostatin antagonist has an opposite effects. The observed phenomenon can underlie the potentiating effect of somatostatin on the dynamics of controlled vagal bradycardia.
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