Abstract
Abstract Glomerular hyperfiltration associated with early diabetes mellitus (DM) is a risk factor for the development of progressive diabetic nephropathy. Though pathogenesis of hyperfiltration is not well-known but has been attributed to glomerular hemodynamic and tubular factors. Renal hyperfiltration due to hemodynamic function abnormalities are seen in animal models of DM, including increased intraglomerular capillary pressure and glomerular hyperfiltration and thought to be due to afferent arteriolar vasodilatation, efferent vasoconstriction, and suppression of tubuloglomerular feedback.
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