Abstract
Introduction Morphological abnormalities of extensive nerve cell death have been mainly observed in cerebral cortex, hippocampus and cerebellum with a long-term use of alcohol. Objectives & Aims We try to to investigate the mechanism of the brain atrophy in alcoholics. Methods We reviewed consecutive CT examinations in 170 patients and 17 controls (from 23 to 85 years old) with symmetrical intracranial ventricular system and normal density of the white and gray matter. Results There was a significant difference (p Conclusions We hypothesized that the spasmodic drinking might be caused by abnormal excitation of neural circuit and the higher the frequency, the more brain atrophy progresses.
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