Abstract
Hepatic ischemia reperfusion injury (HIRI) is a complex pathophysiological process involving multiple factors. It is directly associated with the clinical prognosis. In order to develop reasonable intervention strategies and to improve the prognosis of patients with HIRI, more and more attention has been paid by researchers to investigate the pathophysiological changes and mechanisms of HIRI. A large number of studies have confirmed that the mechanisms of HIRI are involved in metabolic acidosis, calcium overload, oxygen free radicals, apoptosis, endothelin and nitric oxide concentration imbalance, complement, xanthine oxidase, heat shock protein, Kupffer cells, lymphocytes and neutrophils activation and many other factors. It is critical for clinical intervention of HIRI to further explore its mechanism. Key words: Hepatic ischemia reperfusion injury; Complement; Mechanism
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