Abstract
Beta titanium alloys were developed for biomedical applications due to the combination of its mechanical properties including low elasticity modulus, high strength, fatigue resistance, good ductility and with excellent corrosion resistance. With this perspective a metastable beta titanium alloy Ti-12Mo-13Nb was developed with the replacement of both vanadium and aluminum from the traditional alloy Ti-6Al-4V. This paper presents the microstructure, mechanical properties of the Ti-12Mo-13Nb hot swaged and aged at 500 °C for 24 h under high vacuum and then water quenched. The alloy structure was characterized by X-ray diffraction and transmission electron microscopy. Tensile tests were carried out at room temperature. The results show a microstructure consisting of a fine dispersed α phase in a β matrix and good mechanical properties including low elastic modulus. The results indicate that Ti-12Mo-13Nb alloy can be a promising alternative for biomedical application.
Highlights
The increase in life expectancy of the population has led to a greater number of older people[1] and this in turn, has increased the demand for materials suitable to biomedical implants[2]
The XRD results show that after an aging of 24 h at 500 °C, only α precipitates exist in the β matrix of aged Ti-12Mo-13Nb alloy
The results show than the Ti-12Mo-13Nb alloy hot forged and aged at 500 °C for 24 h presented a microstructure consisting of very fine dispersed α phase in a β matrix and with good mechanical properties
Summary
The increase in life expectancy of the population has led to a greater number of older people[1] and this in turn, has increased the demand for materials suitable to biomedical implants[2]. Biomaterials used for replacement implants have to combine excellent biocompatibility with appropriate mechanical properties. The biocompatibility of a metallic implant is directly determined by its corrosion resistance and the biological effects of released metallic ions. These effects are diverse: cytotoxicity (inducing cell or tissue death), genetic damage or immune response[4]. A low value of Young’s modulus is required in order to minimize the modulus mismatch between implant and surrounding bone tissues. This stiffness mismatch can induce the stress shielding phenomenon.
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