Abstract
Estimated glomerular filtration rate (eGFR) has significant heritability, varying from 36% to 75% in different studies.1,2 Genome-wide association studies (GWAS) have now identified over 400 significant loci for eGFR, most of which map to noncoding regions.3,4 A major challenge for the field is now to identify causal genes and cell types for each of these loci to enable mechanistic interpretation of GWAS findings.
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