Abstract
Negative-pressure wound therapy, commercially known as vacuum-assisted closure (V.A.C.®) therapy, has become one of the most popular (and efficacious) interim (prior to flap reconstruction) or definite methods of managing deep sternal wound infection. Complications such as profuse bleeding, which may occur during negative-pressure therapy but not necessarily due to it, are often attributed to a single factor and reported as such. However, despite the wealth of clinical experience internationally available, information regarding certain simple considerations is still lacking. Garnering information on all the factors that could possibly influence the outcome has become more difficult due to a (fortunate) decrease in the incidence of deep sternal wound infection. If more insight is to be gained from fewer clinical cases, then various potentially confounding factors should be fully disclosed before complications can be attributed to the technique itself or improvements to negative-pressure wound therapy for deep sternal wound infection can be accepted as evidence-based and the guidelines for its use adapted. The authors propose the adoption of a simple checklist in such cases.
Highlights
Negative-pressure wound therapy, commercially known as vacuum-assisted closure (V.A.C.®) therapy, has become one of the most popular interim or definite methods of managing deep sternal wound infection
Serious bleeding during topical negative-pressure wound therapy (NPWT), commercially known as vacuumassisted closure (V.A.C.®) therapy, for deep sternal wound infection (DSWI), is exceedingly rare
Two mechanisms have been linked to serious bleeding and NPWT therapy: infectious erosion [5,6] or, in the case of the right ventricle (RV), a combination of mechanics
Summary
Negative-pressure wound therapy, commercially known as vacuum-assisted closure (V.A.C.®) therapy, has become one of the most popular (and efficacious) interim (prior to flap reconstruction) or definite methods of managing deep sternal wound infection. Serious bleeding during topical negative-pressure wound therapy (NPWT), commercially known as vacuumassisted closure (V.A.C.®) therapy, for deep sternal wound infection (DSWI), is exceedingly rare. Two mechanisms have been linked to serious bleeding and NPWT therapy: infectious erosion [5,6] or, in the case of the RV, a combination of mechanics (displacement of the heart towards or in between the sternal edges [7] and fibrous adherence of the RV to the sternum [1]).
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