Abstract
My purpose was to evaluate the hypothesis that the pneumotaxic center could continue to regulate ventilatory activity in deep anesthesia. In decerebrate cats, the respiratory pattern was reversibly altered from eupnea to apneusis by cooling the rostral pons. Such cooling markedly changed phrenic activity even after doses of halothane, pentobarbital, or chloralose-urethane had almost entirely eliminated this activity in eupnea. It is concluded that both pontile and medullary mechanisms control eupneic ventilatory activity in unanesthetized as well as deeply anesthetized preparations.
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