Abstract
It was shown, that modulation of macrophage activity by sodium phthalhydrazide (APH) increases concentration of anti-inflammatory cytokine TGF- $\boldsymbol{\beta}$ in the pancreas and decreases content of the proinflammatory cytokine complex in the pancreas and blood plasma of rats with experimental diabetes mellitus type 2 (DM2). It is possible, that shifts in macrophage production underlie the activation of regenerative processes (increasing of proliferation and decreasing in intensity of apoptosis of pancreatic $\boldsymbol{\beta}$ -cells) at administration of APH.
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