Abstract
It is generally believed that biological effects of ion� izing radiation in irradiated cells are determined by DNA lesions and that DNA breaks induce lowdose radiation effects, such as adaptive response and genomic instability (1). Since natural radioactivity is permanently affecting living creatures, adaptive response has formed in the course of evolution as a mechanism of cell protection against this factor. How� ever, the issues regarding the universality, genotypic determination, and mechanisms of adaptive response as well as the dependence of adaptive response expres� sion on age, environmental conditions, immune sta� tus, and other factors remain to be elucidated (2).
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