Abstract
In children with short stature born small for gestational age (SGA), growth hormone (GH) treatment is associated with an acceleration of growth allowing most to achieve an adult height adequate for their target height. The pathophysiology of growth failure may arise from anomalies in the GH–IGF axis, including deficits in GH/IGF-I concentrations and/or GH/IGF-I sensitivity.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
